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¹Assistant Professor, Aksaray University Medicine Faculty, Departmant of Ophtalmology, Aksaray, Turkey
²Professor, Osmangazi University Medicine Faculty, Department of Ophtalmology, Eskisehir,Turkey
³Associate Professor, Osmangazi University Medicine Faculty, Department of Ophtalmology, Eskisehir,Turkey DOI : 10.37845/ret.vit.2021.30.69 A 28-year old male patient underwent cataract surgery and secondary IOL implantation in the sulcus at an external centre for a diagnosis of traumatic cataract to the right eye. The patient then had reduced vision and was diagnosed with CMO. Despite a total of 15 anti- VEGF applications at the external centre, the edema did not improve. In our examination, it was seen that the IOL had been placed in the sulcus and one leg was suspended on the iridotomy. IOP was 26mmHg. On FFA, there was seen to be leakage in the optic disc, peripheral retina and macula. It was thought that the CMO was secondary to infl ammation caused by contact of the IOL on the iris. Surgery was performed by suturing the haptics to the sclera using the patient?s own IOL. Then no CMO was observed throughout 2 years. The elevated IOP continued and the causes of this were thought to be steroid use, infl ammation and damage at the micro level in the angular structure associated with the trauma. Keywords : cystoid macular edema, etiological factors, Irvine-Gass syndrome, Pseudophakic cystoid macular edema